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Data updated: May 26, 2026

MYCOPHENOLATE MOFETIL HYDROCHLORIDE

Genetic Support
Immunology Approved 2016-10-28

Mycophenolate mofetil is an antimetabolite immunosuppressant indicated for the prophylaxis of organ rejection in adult and pediatric patients (3 months of age and older) receiving allogeneic kidney, heart, or liver transplants. It is utilized as part of a multi-drug regimen in combination with other immunosuppressive agents, such as cyclosporine and corticosteroids, to improve long-term graft survival.

Source: FDA Label • AMNEAL

How MYCOPHENOLATE MOFETIL HYDROCHLORIDE Works

Mycophenolate mofetil is a prodrug that is rapidly hydrolyzed to its active metabolite, mycophenolic acid (MPA). MPA is a selective, uncompetitive, and reversible inhibitor of inosine monophosphate dehydrogenase (IMPDH), which is the rate-limiting enzyme in the *de novo* pathway of guanosine nucleotide synthesis. Because T- and B-lymphocytes rely almost exclusively on *de novo* synthesis for proliferation (unlike other cell types that utilize salvage pathways), MPA effectively blocks their DNA synthesis and cellular division. Beyond lymphocytotoxicity, MPA shifts lymphocytes into an anergic state, suppresses the production of inflammatory cytokines (e.g., IFN-γ, TNF-α), and inhibits the glycosylation of adhesion molecules, thereby reducing the recruitment of immune cells into the allograft.

Source: FDA Label
8
Indications
--
Phase 3 Trials
9
Years on Market

Details

Status
Discontinued
First Approved
2016-10-28
Routes
INJECTION
Dosage Forms
INJECTABLE

MYCOPHENOLATE MOFETIL HYDROCHLORIDE Approval History

2017
2018
2019
2020
2021
2022
2023
2024
2025
2026
Original
New Indication
New Form
Label Update
49 FDA actions from 2016 to 2024
Aug 2024 SUPPL
Update · REMS
Jul 2024 SUPPL
Label · Labeling
May 2024 SUPPL
Label · Labeling

What MYCOPHENOLATE MOFETIL HYDROCHLORIDE Treats

1 indications

MYCOPHENOLATE MOFETIL HYDROCHLORIDE is approved for 1 conditions since its original approval in 2016. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Organ Rejection
Source: FDA Label

MYCOPHENOLATE MOFETIL HYDROCHLORIDE Boxed Warning

EMBRYOFETAL TOXICITY, MALIGNANCIES and SERIOUS INFECTIONS Use during pregnancy is associated with increased risks of first trimester pregnancy loss and congenital malformations. Avoid if safer treatment options are available. Females of reproductive potential must be counseled regarding pregnancy prevention and planning [see Warnings and Precautions (5.1) , Use in Special Populations (8.1 , 8.3) ] . Increased risk of development of lymphoma and other malignancies, particularly of the skin [see W...

MYCOPHENOLATE MOFETIL HYDROCHLORIDE Target & Pathway

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Target

MTOR (Mechanistic Target of Rapamycin) Intracellular Kinase

MYCOPHENOLATE MOFETIL HYDROCHLORIDE Competitive Set

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Active Pipeline

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Key Completed Trials

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Completed studies with published results, ranked by significance

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

MYCOPHENOLATE MOFETIL HYDROCHLORIDE FDA Label Details

Indications & Usage

MYCOPHENOLATE MOFETIL HYDROCHLORIDE is indicated for the treatment of Organ Rejection.

⚠️ BOXED WARNING

WARNING: EMBRYOFETAL TOXICITY, MALIGNANCIES and SERIOUS INFECTIONS Use during pregnancy is associated with increased risks of first trimester pregnancy loss and congenital malformations. Avoid if safer treatment options are available. Females of reproductive potential must be counseled regarding pre...

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.