TheraRadar
Data updated: May 26, 2026

BONCRESA (denosumab-mobz)

RANK Ligand Blocking Activity
Oncology Approved 2025-12-19

Boncresa helps patients with osteoporosis who face a high risk of fractures, including those with a history of broken bones or those who cannot tolerate other therapies. It treats postmenopausal women and men with osteoporosis, as well as individuals who develop bone loss from taking systemic glucocorticoids for at least six months. This medication is also used to increase bone mass in patients receiving certain hormone-blocking therapies for breast or prostate cancer.

Source: FDA Label • AMNEAL PHARMS LLC • RANK Ligand Inhibitor

How BONCRESA Works

Boncresa works by binding to RANKL, a protein that is essential for the formation and survival of osteoclasts, the cells responsible for bone resorption. By preventing RANKL from activating its receptor, the drug inhibits the breakdown of bone and increases overall bone mass and strength.

1
Indication
--
Phase 3 Trials
0
Years on Market

Details

Status
Prescription
First Approved
2025-12-19
Routes
SUBCUTANEOUS
Dosage Forms
INJECTABLE

Companies

Active Ingredient: DENOSUMAB-MOBZ

BONCRESA Approval History

2026
Original
New Indication
New Form
Label Update
1 FDA actions from 2025 to 2025
Dec 2025 ORIGINAL
Update

What BONCRESA Treats

3 indications

BONCRESA is approved for 3 conditions since its original approval in 2025. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Osteoporosis
  • Glucocorticoid-Induced Osteoporosis
  • Breast Cancer
Source: FDA Label

BONCRESA Boxed Warning

SEVERE HYPOCALCEMIA IN PATIENTS WITH ADVANCED KIDNEY DISEASE Patients with advanced chronic kidney disease (eGFR <30 mL/min/1.73 m 2 ), including dialysis-dependent patients, are at greater risk of severe hypocalcemia following denosumab products administration. Severe hypocalcemia resulting in hospitalization, life-threatening events and fatal cases have been reported [see Warnings and Precautions (5.1) ] . The presence of chronic kidney disease-mineral bone disorder (CKD-MBD) markedly increase...

Auto-substitute OK for Prolia

Pharmacists can substitute BONCRESA for Prolia without calling the prescriber. This is a lower-cost alternative with no clinically meaningful differences.

BONCRESA Competitive Set

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Filters applied: drops same-active-ingredient (505(b)(2) reformulations), route-mismatch (topical vs systemic), and cross-therapeutic-area matches in same-indication rings.

What's emerging in BONCRESA's indications

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Phase 3 candidates targeting molecules with no FDA-approved drug, in indications BONCRESA treats. First-in-class if their pivotal trials read out positive.

Drugs Similar to BONCRESA

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Shared indications:
OsteoporosisGlucocorticoid-Induced OsteoporosisBreast Cancer
BOSAYA
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BIOCON BIOLOGICS INC
Shared indications:
OsteoporosisGlucocorticoid-Induced OsteoporosisBreast Cancer
CONEXXENCE
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Key Completed Trials

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

BONCRESA FDA Label Details

Indications & Usage

FDA Label (PDF)

BONCRESA is indicated for the treatment of Osteoporosis; Glucocorticoid-Induced Osteoporosis; Breast Cancer.

⚠️ BOXED WARNING

WARNING: SEVERE HYPOCALCEMIA IN PATIENTS WITH ADVANCED KIDNEY DISEASE Patients with advanced chronic kidney disease (eGFR <30 mL/min/1.73 m 2 ), including dialysis-dependent patients, are at greater risk of severe hypocalcemia following denosumab products administration. Severe hypocalcemia resultin...

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.