TheraRadar
Data updated: May 26, 2026

UNLOXCYT (cosibelimab-ipdl)

Programmed Death Ligand-1 Antagonists Genetic Support
Oncology Approved 2024-12-13

UNLOXCYT is indicated for the treatment of Cutaneous Squamous Cell Carcinoma.

Source: FDA Label • CHECKPOINT THERAPEUTICS INC • Programmed Death Ligand-1 Blocker

How UNLOXCYT Works

Cosibelimab-ipdl works by binding to PD-L1, a protein that can be expressed on tumor cells to inhibit the body's anti-tumor immune response. By blocking the interaction between PD-L1 and its receptors, PD-1 and B7.1, the drug releases the inhibitory effects on T cells, thereby restoring cytotoxic T-cell activity and proliferation. Additionally, the drug has been shown to induce antibody-dependent cell-mediated cytotoxicity (ADCC) in vitro.

2
Indications
--
Phase 3 Trials
1
Years on Market

Details

Status
Prescription
First Approved
2024-12-13
Routes
INJECTION
Dosage Forms
INJECTABLE

Companies

Active Ingredient: COSIBELIMAB-IPDL

UNLOXCYT Approval History

2025
2026
Original
New Indication
New Form
Label Update
2 FDA actions from 2024 to 2025 · 1 indication expansions
Nov 2025 SUPPL
Efficacy
Dec 2024 ORIGINAL
New Drug · Type 1 - New Molecular Entity

What UNLOXCYT Treats

1 indications

UNLOXCYT is approved for 1 conditions since its original approval in 2024. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Cutaneous Squamous Cell Carcinoma
Source: FDA Label

UNLOXCYT Target & Pathway

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Target

PD-1 (Programmed Cell Death Protein 1) Immune Checkpoint

An immune checkpoint receptor on T-cells that acts as an 'off switch' for immune responses. Cancer cells often exploit PD-1 by expressing its ligands (PD-L1/PD-L2), allowing tumors to evade immune detection. Blocking PD-1 releases this brake, enabling T-cells to recognize and attack cancer cells.

Pathway Context

PD-1 on T-cells binds to PD-L1/PD-L2 on tumor cells, suppressing immune attack

PD-L1 (Programmed Death-Ligand 1) ligand

A protein expressed on tumor cells and immune cells that binds to PD-1 on T-cells, suppressing immune responses. Many cancers overexpress PD-L1 to avoid immune attack. Blocking PD-L1 prevents this immune suppression, allowing the body's T-cells to fight cancer.

PD-L2 (Programmed Death-Ligand 2) ligand

A second ligand for PD-1, expressed on antigen-presenting cells and some tumors. PD-L2 binds PD-1 with higher affinity than PD-L1 but is less widely expressed. Drugs blocking PD-1 prevent both PD-L1 and PD-L2 interactions.

UNLOXCYT Competitive Set

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Filters applied: drops same-active-ingredient (505(b)(2) reformulations), route-mismatch (topical vs systemic), and cross-therapeutic-area matches in same-indication rings.

What's emerging in UNLOXCYT's indications

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Phase 3 candidates targeting molecules with no FDA-approved drug, in indications UNLOXCYT treats. First-in-class if their pivotal trials read out positive.

Drugs Similar to UNLOXCYT

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KEYTRUDA
PEMBROLIZUMAB
1 shared
Merck
Shared indications:
Cutaneous Squamous Cell Carcinoma
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Active Pipeline

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Key Completed Trials

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Completed studies with published results, ranked by significance

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

UNLOXCYT FDA Label Details

Indications & Usage

FDA Label (PDF)

UNLOXCYT is indicated for the treatment of Cutaneous Squamous Cell Carcinoma.

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.