TheraRadar
Data updated: May 26, 2026

ZYNYZ (retifanlimab-dlwr)

Programmed Death Receptor-1-directed Antibody Interactions Genetically Validated
Orphan Drug Priority Review Accelerated Approval Fast Track
Oncology Approved 2023-03-22

ZYNYZ is indicated for the treatment of Squamous Cell Carcinoma of the Anal Canal; Merkel Cell Carcinoma.

Source: FDA Label • INCYTE CORP • Programmed Death Receptor-1 Blocking Antibody

How ZYNYZ Works

Retifanlimab-dlwr binds to the PD-1 receptor on T cells, which blocks its interaction with the ligands PD-L1 and PD-L2. Normally, when these ligands bind to the PD-1 receptor, they inhibit T-cell proliferation and cytokine production, allowing some tumors to evade immune surveillance. By blocking this pathway, the drug prevents the inhibition of the immune response and potentiates T-cell activity against tumor cells.

4
Indications
--
Phase 3 Trials
2
Priority Reviews
3
Years on Market

Details

Status
Prescription
First Approved
2023-03-22
Patent Cliff
2030

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Routes
INTRAVENOUS
Dosage Forms
INJECTABLE

Companies

Active Ingredient: RETIFANLIMAB-DLWR

ZYNYZ Approval History

2024
2025
2026
Original
New Indication
New Form
Label Update
5 FDA actions from 2023 to 2026 · 3 indication expansions
Mar 2026 SUPPL
Efficacy
Dec 2025 SUPPL
Efficacy
May 2025 SUPPL Priority
Efficacy

What ZYNYZ Treats

2 indications

ZYNYZ is approved for 2 conditions since its original approval in 2023. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Squamous Cell Carcinoma of the Anal Canal
  • Merkel Cell Carcinoma
Source: FDA Label

ZYNYZ Target & Pathway

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Target

PD-1 (Programmed Cell Death Protein 1) Immune Checkpoint

An immune checkpoint receptor on T-cells that acts as an 'off switch' for immune responses. Cancer cells often exploit PD-1 by expressing its ligands (PD-L1/PD-L2), allowing tumors to evade immune detection. Blocking PD-1 releases this brake, enabling T-cells to recognize and attack cancer cells.

Pathway Context

PD-1 on T-cells binds to PD-L1/PD-L2 on tumor cells, suppressing immune attack

PD-L1 (Programmed Death-Ligand 1) ligand

A protein expressed on tumor cells and immune cells that binds to PD-1 on T-cells, suppressing immune responses. Many cancers overexpress PD-L1 to avoid immune attack. Blocking PD-L1 prevents this immune suppression, allowing the body's T-cells to fight cancer.

PD-L2 (Programmed Death-Ligand 2) ligand

A second ligand for PD-1, expressed on antigen-presenting cells and some tumors. PD-L2 binds PD-1 with higher affinity than PD-L1 but is less widely expressed. Drugs blocking PD-1 prevent both PD-L1 and PD-L2 interactions.

ZYNYZ Competitive Set

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Filters applied: drops same-active-ingredient (505(b)(2) reformulations), route-mismatch (topical vs systemic), and cross-therapeutic-area matches in same-indication rings.

What's emerging in ZYNYZ's indications

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Phase 3 candidates targeting molecules with no FDA-approved drug, in indications ZYNYZ treats. First-in-class if their pivotal trials read out positive.

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KEYTRUDA
PEMBROLIZUMAB
1 shared
Merck
Shared indications:
Merkel Cell Carcinoma
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Active Pipeline

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Key Completed Trials

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

ZYNYZ FDA Label Details

Indications & Usage

FDA Label (PDF)

ZYNYZ is indicated for the treatment of Squamous Cell Carcinoma of the Anal Canal; Merkel Cell Carcinoma.

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.